Acquired long QT syndrome (LQTS) is undeniably more frequently encountered in clinical practice as compared to congenital LQTS [1]. Pharmaceutical drugs are by
LQTS may be either congenital or acquired. Acquired LQTS usually results from drug therapy, although other factors such as hypokalemia, hypomagnesemia, and bradycardia can increase the risk of drug-induced LQTS. The clinical manifestations, diagnosis, and management of acquired LQTS will be reviewed here.
LQTS, and drugs that have been associated with QTP and TdP. Long QT syndrome (LQTS) characterized by prolongation of the QT interval, may occur as
Although there is no cure for LQTS, we can treat the disorder to prevent fainting and dangerous arrhythmias. This form of acquired LQTS is called drug-induced
Traditionally, LQTS is divided into congenital (c-LQTS) and acquired (a-LQTS) forms. Drug-induced LQTS is the most common cause of a-LQTS; as a matter of fact
Patients with LQTS should always avoid drugs that prolong the QT interval (www.crediblemeds.org). In patients with LQTS, it is important to correct electrolyte
Congenital LQTS and Drugs to Avoid List. There are over 290 medicines on the Drugs to Avoid (DTA) list for patients with Congenital LQTS (CLQTS).
Patients with LQTS should always avoid drugs that prolong the QT interval (www.crediblemeds.org). In patients with LQTS, it is important to correct electrolyte
Acquired LQTS may occur secondary to ingestion of QT interval-prolonging drugs, electrolyte imbalances, or bradyarrhythmias. Patients with LQTS
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